FRDA Splicing Researcher: Elevate Frataxin Levels
Università di Trento invites researchers to join a FRDA project investigating how reprogramming of FXN pre-mRNA splicing can upregulate frataxin expression, a mitochondrial protein essential for iron-sulfur cluster biogenesis and cellular energy metabolism.
The program aims to explore the molecular and functional consequences of a novel therapeutic strategy designed to increase endogenous frataxin expression and to advance understanding of Friedreich ataxia through collaborative research efforts.
The program aims to explore the molecular and functional consequences of a novel therapeutic strategy designed to increase endogenous frataxin expression and to advance understanding of Friedreich ataxia through collaborative research efforts.